Archive for the ‘Skin Allergy’ Category

Allergy to Henna tattoos

Sunday, May 2nd, 2010

Summer is here, it’s time for a sunny break abroad and perhaps a temporary tattoo which is very fashionable. But beware, temporary Henna tattoos done on unsuspecting holiday makers in tourist resorts can have a disfiguring long-term and even dangerous result.  The usually harmless Henna-plant tattoo chemicals are often illegally darkened by unscrupulous vendors adding a concentrated hair dye chemical called PPD (para phenylene diamine) or “Black Henna”.  This dangerously concentrated hair dye chemical is highly sensitising when applied neat onto the skin and in 15% of people will lead to a delayed allergic reaction some days later (usually after returning home from their holiday).  Tell-tale allergy signs include irritation and reddening of the tattooed skin, later resulting in a blistering and scarring contact dermatitis which in some cases can last many weeks. The long-term consequences are that once sensitised to PPD, you will have a life-long risk of reacting to other less concentrated-PPD containing hair colourings, black rubber products, sulphonamide antibiotics, local anaesthetics and even sunscreens containing PABA (para-aminobenzoic acid). Therefore be extremely cautious about having any temporary Henna tattoos applied to your skin whilst on holiday.

Reference  http://en.wikipedia.org/wiki/Henna

Skin germ makes eczema worse

Wednesday, April 7th, 2010

The Staphylococcus Aureus (Staph) skin germ plays a pivotal role in aggravating moderate to severe atopic eczema especially in children.  Eczema first occurs in infancy but may persist through childhood into adulthood. Many factors are implicated and certain and promote eczema such as our gene makeup (atopy), skin allergy (to dust mites, pet dander and food allergens). The common skin Staph bacterium carried by us on the skin seems to play an important role in triggering eczema exacerbations.  While 10 and 40% of the general population carry the germ Staph on their skin, a whopping 90% of eczema sufferers have this germ on their skin. Studies reveal that 30% of moderate eczema sufferers are allergic to a bacterial endotoxin produced by the Staph, while in severe and difficult to control eczema, up to 68% of children will have an allergy to this germ. The Staph also seems to make established eczema more difficult to treat by increasing skin resistance to therapeutic steroid creams – they just don’t seem to work as well if there is a Staph infection present.

Studies show that systematically eradicating the Staph from the skin will make the eczema easier to control and  ultimately clear. In an established infection, the eczema will spread rapidly appearing bright red, crusty, weepy and blistered. The Staph can also persist by being harboured in the nasal passages, under dirty finger nails and in moist skin creases. Fortunately most Staph will respond to antibiotic skin creams such as Mupirocin (Naseptin), Fusidic acid (Fucidin) as well as to antiseptic skin disinfectants such as povidine iodine (Betadine), hydrogen peroxide, potassium permanganate and chlorhexidine (Hibitane). The beneficial effect of using sodium hypochlorite (found in bleech) and silver impregnated garments has not been proven.  In established Staph infections presenting with Impetigo (skin honey crusts), Folliculitis (pimples and pustules) and Cellulitis (widespread skin surface infection), oral antibiotics such as Flucloxacillin, Erythromycin and Co-amoxyclav will effectively treat the skin infection (unless the superbug MRSA (Methicillin Resistant Staphylococcus Aureus) is present). Read more about managing eczema

Skin infection causes eczema

Thursday, February 4th, 2010

The Staphylococcus Aureus (Staph) skin germ plays a pivotal role in aggravating moderate to severe atopic eczema especially in children.  Eczema first occurs in infancy but may persist through childhood into adulthood. Many factors have been implicated in causing and promoting eczema such as our gene makeup (atopy), skin allergy (to dust mites, pet dander and food allergens) and skin damage. The common skin Staph bacterium carried on our skin plays an important role in triggering eczema exacerbations.  While 10 and 40% of the general population naturally carry Staph on their skin, a whopping 90% of eczema sufferers have this germ inhabiting their skin. Studies reveal that 30% of moderate eczema sufferers are actually allergic to a toxin produced by the Staph, while in severe or difficult to control eczema, up to 68% of sufferers will have an allergy to it. The Staph also seems to make established eczema more difficult to treat by increasing skin resistance to therapeutic steroid creams – they just don’t seem to work as well if there is a Staph infection present.

Studies show that systematically eradicating the Staph from the skin will make the eczema easier to control and ultimately clear. In an established infection, the eczema will spread rapidly appearing bright red, crusty, weepy and blistered. The Staph can also persist by being harboured in the nasal passages, under dirty finger nails and in moist skin creases. Fortunately most Staph will be eliminated by antibiotic skin creams such as Mupirocin (Naseptin) or Fusidic acid (Fucidin) as well as by antiseptic skin disinfectants such as povidine iodine (Betadine), hydrogen peroxide, potassium permanganate and chlorhexidine (Hibitane). The beneficial effect of using sodium hypochlorite (found in bleech) and silver impregnated garments has not been proven.  In more extensive Staph infections presenting with Impetigo (skin honey crusts), Folliculitis (pimples and pustules) and Cellulitis (widespread skin surface infection), oral antibiotics such as Flucloxacillin, Erythromycin and Co-amoxyclav will effectively treat the skin infection (unless the superbug MRSA (Methicillin Resistant Staphylococcus Aureus) is present).

http://pediatrics.aappublications.org/cgi/content/abstract/120/Supplement_3/S122